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Another mechanism by which the endo/lysosomal compartment can influence APAP-induced hepatotoxicity is through the clearance of damaged mitochondria in a process also called mitophagy, consisting in the degradation of dysfunctional mitochondria by lysosomal proteases upon fusion of the autophagosomes-containing mitochondria with lysosomes, which is discussed in the next section

I think that's a good empirical way of thinking about it

Rare Disease Advisor Chae Y, Kwon SH, Nam JH, et al

An instructive example of how ipamorelin effects and dosing interact comes from the bone study by Svensson et al., 2000

GLP-1: from lizard venom to modern medicine One of the most fascinating origin stories in pharmacology

These findings open that door, he said