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Consequently, a decrease in PKA-pCREB levels may contribute to impairment of synaptic plasticity and learning function in AD (Gong et al., 2006

doi:10.1007/s12539-009-0059-0

Following an acquired brain injury, acute glutamate release induces excitotoxicity, neuronal injury, cell death, impairing the function and metabolism of surviving neurons and potentiating KP dysregulation [251, 252]