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In various experimental animal models, the use of neutrophil elastase inhibitor has been shown to reduce the incidence and severity of collagen-induced arthritis (CIA) and mitigate histologically demonstrated damage to articular cartilage (24, 95)

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However, very limited information is available about the exact metabolic functions of disease-induced GST isoenzymes and about their endogenous substrates

2015;6 doi: 10.1038/ncomms10112