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The exploited pancreatic pre-neoplastic lesion organoid transplantion in syngeneic mouse model resulted a good approximation of human PDAC pathology, which develops slowly together with desmoplasia establishment [46, 56, 57]

CCDS1, caused by genetic variants in the SLC6A8 gene, is an X-linked inherited metabolic disorder with common clinical manifestations including mental retardation, severe speech disorders, epilepsy, movement disorders, and behavioral disorders

The body composition changes they produce are similar to what happens with any significant weight loss