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In mammals, aging of the immune compartment has been associated with increased DNA damage, to a shift in immune cell pools toward more myeloid over lymphoid cells (myeloid shift) and to an increased secretion of inflammatory mediators 6

TB-500 appears to sequester G-actin, potentially influencing cytoskeletal reorganization and cellular motility in vitro

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This is the formidable challenge that traditional treatments struggle to overcome, and it's precisely where the potential of a systemic agent like BPC 157 becomes so compelling