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The Journal of biological chemistry 268 , 1965019655 (1993)

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The rationale for this repurposing lies in the profound pathophysiological overlap between T2DM and PCOS: given that IR is a primary driver of ovarian androgen excess, agents that powerfully correct systemic metabolic dysfunction are hypothesized to indirectly restore ovarian homeostasis

If matching the actual study protocols matters to you, thats the one most of the literature was built on